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Moreover, because GLP-1R is expressed throughout the body, stimulation with a GLP-1R agonist or indirectly with a DPP-IV inhibitor can have a broad systemic effect on systemic metabolism, which, in turn, ameliorates peripheral and central insulin resistance in AD and MD (Athauda and Foltynie, 2016)

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In tumor cells, evasion of ferroptosis mediated by oncogenes or carcinogenic signaling contributes to tumor onset, progression, metastasis and resistance to treatment