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Proteomic studies further confirmed that ApoE3 improves cerebrovascular function by inducing extracellular matrix-stabilizing proteins such as Timp3, whereas ApoE4 exacerbates pathological progression by suppressing lipoprotein metabolism-related proteins and activating the complement system [88, 91]
The compound should be avoided during these periods unless specifically recommended by a qualified healthcare provider after careful risk-benefit assessment
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