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To further investigate the relative contribution of glucose-derived carbon (via pyruvate) and fatty-acid derived carbon (from acetyl-CoA) to overall TCA flux and subsequent OXPHOS and ATP generation in ErbB2+ cells with and without Cpt1a, we employed specific inhibitorsUK-5099, targeting MPC, and Etomoxir, which inhibits Cpt1a

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It has since been rebranded as WW Clinic

Previous studies confirmed that lactate was required for fibroblast activation in a pH-dependent manner [13]

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