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Importantly, neuronal survival is continuously stimulated by trophic factors that function through specific receptors that are regulated by MEK/ERK and that lead to c-AMP response element binding protein (CREB) activation, a key regulator of several cellular processes (Wang et al., 2003)

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Each class interacts differently with GLP-1 medications, and the potential for synergistic immunosuppression or altered efficacy varies based on the specific RA regimen