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Who Is Most at Risk for B12 Deficiency

GHK-Cus primary mechanisms include collagen stimulation (Type I and III synthesis increases of up to 70% in vitro), matrix metalloproteinase regulation (simultaneously activating MMPs for damaged matrix removal while upregulating TIMP-1/TIMP-2 for controlled remodeling), wound contraction promotion, and anti-inflammatory NF-B pathway modulation

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