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Clinical trials involving approximately 900 participants found that the adverse event profile was indistinguishable from placebo for most parameters

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[36] Antibodies to intrinsic factor and parietal cells cause the destruction of the oxyntic gastric mucosa, in which the parietal cells are located, leading to the subsequent loss of intrinsic factor synthesis

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Al examined the effects of topical application and intraperitoneal application of the BPC 157 on burn-wound healing burn-gastric lesions

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Rather than simply increasing or decreasing NO production, studies indicate that the compound modulates NO activity contextually, supporting vasodilation and blood flow where healing requires it while counteracting NO-mediated tissue damage in inflammatory contexts (3)

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