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These facts paved the way for the development of a GIP receptor agonist-based therapy for T2DM, looking also for the possibility of finding a dual GLP-1/GIP receptor agonist [17]
In one study, researchers found that IGF-1 causes the proliferation of myofibroblast in a dose-dependent manner
doi: 10.1001/jama.2016.10260 63 JorsalAKistorpCHolmagerPTougaardRSNielsenRHnselmannAet al
2006 Dec;25(6):886-96
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