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In humans, CCK dose-dependently presents early satiety and reduces the energy intake at a buffet style meal, which was attenuated by the CCK-1 antagonist, loxiglumide (107)

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Elevated PARP-1 levels not only help against DNA damage, but are also exploited by EBV, as PARP1 acts as a barrier to productive lytic reactivation (229) and promotes the transition of EBV into the LMP1-induced latency state (230)