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The hyperglycemia-related increased proximal tubular sodium reabsorption results in lower sodium levels at the level of the macula densa increasing GFR via inhibiting TGF, further augmenting the amount of filtered and reabsorbed sodium with increased ATP demands

[4] These metabolites remain detectable for varying windows depending on the route and dose of administration

That means the most beneficial treatments should address a variety of issues simultaneously

Investigation 124 (10), 44734488

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