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This is according to a small study by the Fralin Biomedical Research Institute at VTC

We therefore hypothesise that GLP-1R-dependent phosphorylation of MIC19 at ERMCSs via the VAPB/SPHKAP/PKA pathway might not only highlight a direct role for the GLP-1R in the control of mitochondrial cristae architecture and dynamics, but also in the regulation of lipid precursor transfer across ERMCSs to sustain mitochondrial turnover