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The present study provided evidence that GLP-1R is associated with ASM cell proliferation and the secretion of inflammatory cytokines

Although, any study has not related the viral infection, GSH intracellular levels, and excitotoxic neurodegeneration, the literature suggested that antioxidant responses can prevent the neuron death directly or indirectly

The tendency for some individuals to have received DPP-4is before switching to GLP-1 RAs may account for the longer diabetes duration and the slightly higher prevalence of diabetic complications and hypertension in the GLP-1 RA group

PMCID: PMC8865914

When a GLP-1 agonist activates these receptors, it triggers multiple intracellular signaling cascades including the cAMP/PKA and PI3K/Akt pathways that promote neuronal survival, reduce oxidative stress, suppress inflammation, and improve vascular integrity