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Therefore, in this review we detail the direct and indirect molecular mechanisms by which incretin receptor agonists inhibit fibrotic signalling pathways, and critically appraise the evidence for their preclinical and clinical efficacy in fibrotic disorders across diverse tissues, including the liver, heart, kidney, lung, and joints (Table 1
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The study identifies the regulation of glycolysis as a new mechanism by which PTBP1 crotonylation drives disease progression
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