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This impairment of protective functions aligns with the persistent inflammatory state and elevated cardiovascular risk characteristic of this patient population [56]
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Interestingly, the incubation with the antioxidant N-acetylcysteine (NAC) upon treatment with NP4 resulted in reduced levels of apoptosis, suggestive that the cytotoxicity of NP4 is related to intracellular redox homeostasis (Supplementary Fig
Physical stress and catecholamine release
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