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In line with this, mice with an -cell specific knock-down of PC1/3, resulting in significantly reduced islet GLP-1 content, were able to maintain normal glucose tolerance, suggesting that islet GLP-1 may not be essential and/or that it can be compensated for by intestinally derived GLP-1 under normal conditions

Diabetes Care 2007

Induction of the mitochondrial ndufa4l2 protein by hif-1 decreases oxygen consumption by inhibiting complex i activity

GLP-1Rs are coupled predominantly to G s -proteins and increase cAMP signaling when activated 49,50

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