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[10] These studies should be described as preclinical cytoprotection research, not as evidence for human gastrointestinal treatment
The metabolic inhibitor sodium azide also significantly reduced the cellular uptake of the nanocarriers, suggesting an active cellular process (Fig
The heavy subunit catalyzes formation of -glutamylcysteine, whereas the light subunit increases affinity for glutamate and decreases the inhibition by glutathione (Seelig et al., 1984
the effects were additive, not redundant
However, results may vary from person to person
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