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We hypothesized that TEAS activated cutaneous sensory nerve fibres to express sensory CGRP and SP, and that SP bonded to neurokinin-1 receptor (NK-1R) situated in MCs to initiate MC release of tryptase and 5-HT
Studies of the von Willebrand factor and fibrinogen (important vascular determinants of increased platelet aggregation and secretion that may be involved in the development of thrombosis) also favor the theory of platelet hyperactivity
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