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They are designed to withstand pressure surges and contain ammunition safely
However, the therapeutic utility of doxorubicin is undermined by its dose-dependent cardiotoxicity, possibly arising as a result of futile redox cycling, with NADH dehydrogenase within the mitochondria proposed as the most likely site of anthracycline reduction 9,10
Whether its GLP-1 receptor agonists or the new generation of GLP-1 therapies, one thing remains critical
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