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2019 May;182:144-155
One of the interactions they identified involved the cytosolic form of the selenoprotein glutathione peroxidase, GPX1, which bound strongly to the inactive C145A M pro mutant
How is the FDA going to prioritize follow-up actions, if any, in response to those responses? This article is exclusive to STAT+ subscribers Unlock this article plus in-depth analysis, newsletters, premium events, and news alerts
Peptide hormone exendin-4 stimulates subventricular zone neurogenesis in the adult rodent brain and induces recovery in an animal model of Parkinsons disease
Vascular disease may affect your life in big and small ways
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