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FOXO4-DRI interrupts the interaction between FOXO4 and p53, destabilizing the survival mechanisms of senescent cells

Without a spatially restricted and experimentally identifiable origin of ferroptotic stress, ordered patterns of cell death could simply reflect pre-existing gradients in ferroptosis susceptibility within heterogeneous microenvironments rather than true directional propagation

Multi-agonist therapies combining GLP-1 with GIP or glucagon may amplify these benefits by improving fat metabolism and reducing cardiovascular risk even further