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This, in turn, allows the tendon cells to grow and move during injury repair, speeding up the process [R, R]
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and activating a variety of pathways, including Nrf2, AMPK, SIRT1and FOXO3a
Increasing GSH and/or GSH-dependent antioxidant enzymes directly Many studies have concentrated on the direct effects of synthetic drugs on GSH and GSH-dependent antioxidant enzymes in damaged myocardium without further mechanistic investigations
J Neuropathol Exp Neurol 49:591599
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