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Biomedicine & Pharmacotherapy , 57(34), 145155
According to a study conducted by D
Under oxidative stress, excessive ROS such as OH directly attack DNA, causing single-strand or double-strand breaks, activating PARP-1, consuming NAD and ATP, and generating poly(ADP-ribose), resulting in mitochondrial membrane potential loss and the release of apoptosis-inducing factor, ultimately resulting in cell death [252]
TdGSH prevented LOsG-induced FoxO1 loss
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