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Dihydroxyacetone suppresses mTOR nutrient signaling and induces mitochondrial stress in liver cells
The progression of GC can be observed through various stages, starting from superficial gastritis, then progressing to atrophic gastritis, intestinal metaplasia, gastric epithelial dysplasia, and finally gastric cancer
96 , 97S (1991)
How the interplay between the commensal microbiota, gut barrier integrity, and mucosal immunity regulates brain autoimmunity
1c,d) and was previously suggested as a candidate for a potential activator site, first, because its positive charge might stabilize or bind GSH as GS , and, second, because of its evolutionary conserved position that is equivalent to the GSH-interacting catalytic residue of glutathione transferases 3
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