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At therapeutic doses, GLP-1 drugs cross the blood-brain barrier and dampen dopamine signaling in the brain's core reward center, making addictive substances less rewarding

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Data from preclinical studies suggest that locally produced GLP-1 in the taste bud cells acts as an ancillary but functionally important neurotransmitter in the initial coding of taste information and that it is specifically involved in the perception of sweet [2, 9,10,11,12]

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